Basic Principles of Peripheral Nerve Disorders by S. Rayegani

By S. Rayegani

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H. M. (1986). Selective expression of endogenous lactose-binding lectins and lactoseries glycoconjugates in subsets of rat sensory neurons. 7, (April 1986), pp. ; Ishikawa, Y. & Inoue, S. (1999). Enhanced neural regeneration from transected vagus nerve terminal in diabetic mice in vitro. 5, (April 1999), pp. ; Ishigatsubo, Y. & Ishikawa, Y. (2002). Trachea enhances neurite regeneration from adult rat nodose ganglia in vitro. 16, (March 2002), pp. E. G. (1983). Neurotransmitter candidates of somatosensory primary afferent fibers.

Fig. 3. tv) Electrodiagnostic Medicine Consultation in Peripheral Nerve Disorders 21 Fig. 4. Mixed type of conduction block and demyelination in a patient with carpal tunnel syndrome(From the author personal archive). Fig. 5. complete axonal damage with absence of response in distal and proximal stimulation at the site of injury (From the author personal archive) . 22 Basic Principles of Peripheral Nerve Disorders In neurapraxia with segmental demyelination the nerve fiber axons are intact and no axonal degeneration and nerve destruction occurs.

Fig. 3. Three principal subgroups of adult DRG neurons. Large neurons, immunoreactive for 200 kDa neurofilament, are known to possess large myelinated fibers. Small neurons are divided into peptidergic and non-peptidergic neurons; peptidergic neurons are immunoreactive for CGRP and high affinity NGF receptor trkA, whereas non-peptidergic neurons bind the lectin IB4 and express GDNF receptors. Both groups of small neurons are known to possess small myelinated and unmyelinated fibers (modified from McMahon & Benette, Molecular Basis of Pain Induction, 2000, pp.

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